Pyrimethamine inhibits adult polycystic kidney disease by modulating STAT signaling pathways.

نویسندگان

  • Ayumi Takakura
  • Erik A Nelson
  • Nadeem Haque
  • Benjamin D Humphreys
  • Kambiz Zandi-Nejad
  • David A Frank
  • Jing Zhou
چکیده

Autosomal dominant polycystic kidney disease (ADPKD) is a commonly inherited disorder mostly caused by mutations in PKD1, encoding polycystin-1 (PC1). The disease is characterized by development and growth of epithelium-lined cyst in both kidneys, often leading to renal failure. There is no specific treatment for this disease. Here, we report a sustained activation of the transcription factor signal transducer and activator of transcription 3 (STAT3) in ischemic injured and uninjured Pkd1 knockout polycystic kidneys and in human ADPKD kidneys. Through a chemical library screen, we identified the anti-parasitic compound pyrimethamine as an inhibitor of STAT3 function. Treatment with pyrimethamine decreases cell proliferation in human ADPKD cells and blocks renal cyst formation in an adult and a neonatal PKD mouse model. Moreover, we demonstrated that a specific STAT3 inhibitor, S3I-201, reduces cyst formation and growth in a neonatal PKD mouse model. Our results suggest that PC1 acts as a negative regulator of STAT3 and that blocking STAT3 signaling with pyrimethamine or similar drugs may be an attractive therapy for human ADPKD.

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عنوان ژورنال:
  • Human molecular genetics

دوره 20 21  شماره 

صفحات  -

تاریخ انتشار 2011